In addition, a number of evidences help the role of the genetic purposeful versions of dopamine-inactivating enzymes [sixty,61] and of D2 receptors  in modulating the DMN. In addition, it must also be regarded that alterations of the cerebral vasculature reactivity could also play a role in altering our ability to exploit the blood-oxygenation-level-dependent fMRI signal alterations to detect
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